By Roberto Maroldi, Piero Nicolai, W. Draf
Stroke is the 3rd prime reason for loss of life and carotid atherosclerosis is the best reason for embolic stroke. lately, the necessity for a extra special research of atherosclerotic plaques has been under pressure. info past the measure of narrowing of the vessel lumen appears to be like fascinating and there's massive call for for diagnostic tactics that particularly establish rupture-prone, susceptible plaques because the such a lot widespread reason for surprising ischemic occasions.
This atlas is an updated reference paintings on imaging in carotid artery stenosis written by means of the world over renewed specialists. The authors take the reader step-by-step via illustrated descriptions of state of the art imaging options that they have got helped to strengthen, and display that those concepts are an important within the administration of sufferers. This e-book covers all aspects of imaging in carotid artery stenosis and offers an outlook to destiny features. The "take-home-messages" on the finish of every bankruptcy are a brief assist in day-by-day practice.
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Extra resources for Imaging in Treatment Planning for Sinonasal Diseases
Sample text
With this mechanism, called the “piggy bank” function, the nose is able to recover about 100 ml of water everyday. Nevertheless, during nasal breath- D. Tomenzoli 32 ing at room temperature the daily total loss is about 500 ml of water and 300 kcal (Ingelstedt and Toremalm 1961). 9 Resonance Even though it can not be considered a vital function, the nose acts as a resonance box which gives its contribution, together with paranasal sinuses and pharynx, to the characterization of the tone of the voice.
No sign of brain edema. Lamino papyraces (opposite arrrows) Neoplastic Invasion of Bone, the Orbit and Dural Layers: Basic and Advanced CT and MR Findings a 45 b Fig. 18a,b. Adenocarcinoma of the ethmoid sinus, Gd-enhanced T1. a Tumor and the enhanced dura have similar signal intensity. The thickened dura is seen investing the planum ethmoidalis posteriorly to neoplastic invasion (arrow). b Intracranial intradural spread without brain invasion (arrows) a b Fig. 19a,b. Recurrent adenoid cystic carcinoma, left maxillary sinus was the primary site, Gd-enhanced GE-T1 sequences.
B No sign of dural layer trespassing at this level, bone has been eroded (black arrows). Mild dural thickening (1). c–e On the sagittal planes, bone erosion and subtotal replacement of the hyperintense signal of the focally thickened dura suggest intracranial extradural invasion. Posterior sphenoid sinus wall destruction with hypophysis invasion is present. Blockage of frontal sinus drainage is associated with mucous retention and mucosal thickening Fig. 17. Sinonasal undifferentiated carcinoma, Gd-enhanced T1.