By Collin, Catherine
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Reactive oxygen species and the central nervous system, J. Neurochem. 59, 1609-1614. Halliwell, B. and Gutteridge, J. M. C. (1989). Free Radicals in Biology and Medicine. Clarendon Press, Oxford. Herkenham, M. (1987). Mismatches between neurotransmitters and receptor localizations in brain: observations and implications, Neuroscience 23, 1-38. Hornykeiwicz, O. (1966). Dopamine and brain function, Pharmacol Rev. 18, 925-964. Hughes, A. J. D. , Kilford, L. and Lees, A. J. (1992). Accuracy of clinical diagnosis of idiopathic Parkinson's disease: a clinico-pathological study of 100 cases, J.
Both folate and vitamin B12 deficiency may cause similar neurological and psychiatric disturbances, including depression, dementia, myelopathy and peripheral neuropathy. SAMe has a variety of pharmacological effects in the CNS, especially on monoamine neurotransmitter metabolism and receptor systems. SAMe has antidepressant properties, and preliminary studies indicate that it may improve cognitive function in patients with dementia. Treatment with methyl donors (betaine, methionine and SAMe) is associated with remyelination in patients with inborn errors of folate and C-l (one-carbon) metabolism.
In all the determinations, the phagocytic cells were isolated at 09:00 h. 7 pg/ml and 7 pg/ml in the stressed animal group. All determinations were accompanied by a control sample free from hormone. Corticostewne 4-pregnene-ll,21-diol-3,20-dione (Sigma) was prepared in PBS solution, starting from a base solution of 100 mg/100 ml, which was followed by diluting working solutions to 2 /xg/ml and 20 /xg/ml for the basal animals and to 5 ixglwX and 50 fig/ml for the stressed animal group. All determinations were accompanied by a control sample free from hormone.